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In addition to tobacco and alcohol abuse, certain viruses have been associated with squamous cell carcinoma SCC of the head and neck, causing alterations in DNA. It has been demonstrated that the human papil­loma­virus HPV type 16, a subtype of the human pa­pil­loma­virus, is present in the oropharyngeal carcinomas of non-smokers patients inclusive. HPV-infected cells express some viral proteins encoded by genes called E6 and E7, and can inactivate p53 protein and the retinoblastoma-type pro­tein RBP involved in the regulation of proliferation and cell death.

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Corresponding author: Dr. Maria Alexandra Barbu, e-mail: m. There is a strong link between the presence of human papillomavirus HPV cervical cancer and its precursors. All patients diagnosed with invasive cervical carcinoma should be evaluated clinically by vaginal exam and rectal imaging by computed tomography, magnetic resonance imaging or PET-CT.

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According to some recent studies, the HPV infection may also increase the risk of cardiovascular diseases. Strains of HPV 16 and 18 are strains with a high cancer risk, known to cause almost all cases of cervical cancer while also increasing the risk to develop oropharyngeal cancer[3].

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One unprotected sexual contact can facilitate contracting of one or more sexually transmitted infections. Sexual transmission vaginal, oral, and anal of HPV is well known. In the first years follow the onset of sexual life, the risk of HPV infection through unprotected sexual contacts is very high. The objective of this study was to establish the HPV genotypes prevalence in unprotected sexual contact transmission.

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